Head injury management pdf
Thurman D, Guerrero J. Nwachuku EL, Puccio AM, Adeboye A, Chang YF, Kim J, Okonkwo DO. Cerebral salt wasting syndrome in brain injury patients: a potential cause of hyponatremia. She subsequently died. Mayers l. Return-to-Play Criteria after Athletic Concussion. This CT scan shows obvious medial bifrontal atrophy. The relation between acute physiological variables and outcome on the Glasgow Outcome Scale and Disability Rating Scale following severe traumatic brain injury. Rutgers DR, Toulgoat F, Cazejust J, Fillard P, Lasjaunias P, Ducreux D. White matter abnormalities in mild traumatic brain injury: a diffusion tensor imaging study. Ascending meningitis secondary to traumatic cerebrospinal fluid leaks. Fontaine A, Azouvi P, Remy P, et al. Hutchison M, Comper P, Mainwaring L, Richards D. The influence of musculoskeletal injury on cognition: implications for concussion research. Genetic influences on outcome following traumatic brain injury.
A post-hoc 2-year follow-up demonstrated increased mortality in those receiving albumin as opposed to saline.Although hypothermic therapy initially appeared promising, and despite the fact that hypothermia decreases intracranial pressure, a large randomized study of 392 patients with head injuries recently demonstrated that hypothermic therapy does not improve outcomes. Levin HS, Williams DH, Eisenberg HM, et al. Altered expression of novel genes in the cerebral cortex following experimental brain injury.
Sakellaris G, Nasis G, Kotsiou M, Tamiolaki M, Charissis G, Evangeliou A. Dexanabinol (HU-211) in the treatment of severe closed head injury: a randomized, placebo-controlled, phase II clinical trial. Thurman DJ, Alverson C, Dunn KA, et al. 0000151368 00000 n
Smith-Bindman R, McCulloch CE, Ding A, Quale C, Chu PW. Older Age Results in Differential Gene Expression after Mild Traumatic Brain Injury and Is Linked to Imaging Differences at Acute Follow-up.
Compared to warfarin, direct oral anticoagulants are associated with lower mortality in patients with blunt traumatic intracranial hemorrhage: A TQIP study. Factors affecting excitatory amino acid release following severe human head injury. APOE epsilon4 allele and amyloid beta-protein deposition in long term survivors of head injury. This 40-year-old woman was anticoagulated with warfarin (Coumadin) and fell out of her hospital bed.
Which On-field Signs/Symptoms Predict Protracted Recovery From Sport-Related Concussion Among High School Football Players?.
Out-of-hospital endotracheal intubation and outcome after traumatic brain injury. Assessment of risk in a series of 610 patients.
He had left hemiparesis. Effect of eliminating compensation for pain and suffering on the outcome of insurance claims for whiplash injury. Li ZM, Xiao YL, Zhu JX, Geng FY, Guo CJ, Chong ZL, et al. Evidence for cellular damage in normal-appearing white matter correlates with injury severity in patients following traumatic brain injury: A magnetic resonance spectroscopy study. Levetiracetam Prophylaxis for Post-traumatic Brain Injury Seizures is Ineffective: A Propensity Score Analysis.
Diffusion Tensor Imaging: A Possible Biomarker in Severe Traumatic Brain Injury and Aneurysmal Subarachnoid Hemorrhage?. m.sc. However, the calcium channel blocker nimodipine was successful in reducing rates of death and severe disability when instituted acutely in patients with head injuries and traumatic subarachnoid hemorrhages, despite its failure to improve outcomes in 2 large trials of patients with all types of traumatic intracranial injuries.Although numerous synthetic neuroprotective agents are under development, several existing substances have shown promise, but other agents have been disappointing.Because of its excitotoxic blocking properties, magnesium chloride has been used to reduce cortical injury in experimentally brain-injured rats. Although some studies have shown favorable long-term outcomes with this procedureThe operative and nonoperative management of intracranial injuries is an ever-evolving area of study and, at present, more a matter of neurosurgical judgment than hard and fast decision rules.In the acute setting, a consultation with a neurosurgeon is critical for patients with moderate or severe head injuries, focal neurological findings, or intracranial pathology identified on neuroimaging.In the acute setting, nasogastric feedings may need to be initiated for patients with significant head injuries and depressed levels of consciousness or dysphagia. Harrison-Felix C, Pretz C, Hammond FM, Cuthbert JP, Bell J, Corrigan J, et al. Bilgic B, Baral-Kulaksizoglu I, Hanagasi H, et al. One study of 17 patients with traumatic brain injuries showed improved motor tone and decreased muscle spasms with intrathecal baclofen, but whether these benefits will translate into improved functioning remains unknown.Botulinum toxin also has shown promise in decreasing hypertonia in patients with head injuries, primarily by improving passive range of motion rather than by decreasing functional disability.Solid data on cognitive enhancing medications for patients with head injury are lacking.
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